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  4. Gαi1 inhibition mechanism of ATP-bound adenylyl cyclase type 5
 
research article

Gαi1 inhibition mechanism of ATP-bound adenylyl cyclase type 5

Narzi, Daniele  
•
van Keulen, Siri Camee  
•
Röthlisberger, Ursula  
January 25, 2021
PLOS ONE

Conversion of adenosine triphosphate (ATP) to the second messenger cyclic adenosine monophosphate (cAMP) is an essential reaction mechanism that takes place in eukaryotes, triggering a variety of signal transduction pathways. ATP conversion is catalyzed by the enzyme adenylyl cyclase (AC), which can be regulated by binding inhibitory, Gαi, and stimulatory, Gαs subunits. In the past twenty years, several crystal structures of AC in isolated form and complexed to Gαs subunits have been resolved. Nevertheless, the molecular basis of the inhibition mechanism of AC, induced by Gαi, is still far from being fully understood. Here, classical molecular dynamics simulations of the isolated holo AC protein type 5 and the holo binary complex AC5:Gαi have been analyzed to investigate the conformational impact of Gαi association on ATP-bound AC5. The results show that Gαi appears to inhibit the activity of AC5 by preventing the formation of a reactive ATP conformation.

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Type
research article
DOI
10.1371/journal.pone.0245197
Author(s)
Narzi, Daniele  
van Keulen, Siri Camee  
Röthlisberger, Ursula  
Date Issued

2021-01-25

Published in
PLOS ONE
Volume

16

Issue

1

Article Number

e0245197

Editorial or Peer reviewed

REVIEWED

Written at

EPFL

EPFL units
LCBC  
Available on Infoscience
February 19, 2021
Use this identifier to reference this record
https://infoscience.epfl.ch/handle/20.500.14299/175384
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