Jalil, AntoinePilot, ThomasBourgeois, ThibautLaubriet, AlineLi, XiaoxuDiedisheim, MarcDeckert, ValérieMagnani, CharlèneLe Guern, Naïgde Barros, Jean-Paul Paı̈sNguyen, MaximePallot, GVouilloz, AdrienProukhnitzky, LilHermetet, FrançoisAires, VirginieLesniewska, ÉricLagrost, LaurentAuwerx, JohanLe Goff, WilfriedVenteclef, NicolasSteinmetz, ÉricThomas, CharlesMasson, David2025-05-132025-05-132025-05-122025-0510.1016/j.xcrm.2025.102131https://infoscience.epfl.ch/handle/20.500.14299/250095Highlights • LPCAT3 and ELOVL5 coordinate PUFA metabolism in macrophages • Dual LPCAT3/ELOVL5 deletion increases membrane cholesterol-enriched domains • This alteration enhances macrophage sensitivity to cytotoxic oxysterolsenPlasmalogen remodeling modulates macrophage response to cytotoxic oxysterols and atherosclerotic plaque vulnerabilitytext::journal::journal article::research article