Notice détaillée
Titre
UPMEYLAN
Formal Name (French)
Molecular Mechanisms of Lung Cancer Development Laboratory
Formal Name (English)
Molecular Mechanisms of Lung Cancer Development Laboratory
Lab Manager
Oates, Andrew Charles
Group ID
U12436
Auteurs affilié
Ancey, Pierre-Benoit Bernard
Contat, Caroline Anne-Lyse
Faget, Julien Robert Rémi
Groeneveld, Svenja Johanna
Kopparam, Jawahar
Masin, Mark Jonathan
Meylan, Etienne
Pascual, Justine
Sabatino, Silvia
Vazquez, Jessica Christine
Contat, Caroline Anne-Lyse
Faget, Julien Robert Rémi
Groeneveld, Svenja Johanna
Kopparam, Jawahar
Masin, Mark Jonathan
Meylan, Etienne
Pascual, Justine
Sabatino, Silvia
Vazquez, Jessica Christine
Institut
ISREC
Faculté
SV
Note
Members of UPMEYLAN-unit
Lien extérieur
http://meylan-lab.epfl.ch
Publications
Caspase-2-mediated cleavage of Mdm2 creates a p53-induced positive feedback loop
Metabolic plasticity and glucose transporter redundancy dictate lung adenocarcinoma growth
Mutant CTNNB1 and histological heterogeneity define metabolic subtypes of hepatoblastoma
Neutrophils and Snail Orchestrate the Establishment of a Pro-tumor Microenvironment in Lung Cancer
Nonstructural protein 3-4A: the Swiss army knife of hepatitis C virus
Overcoming microenvironmental resistance to PD-1 blockade in genetically engineered lung cancer models
RIP off STAT3 to counteract tumor progression
Regulation of Monocyte Functional Heterogeneity by miR-146a and Relb
Response and resistance to NF-κB inhibitors in mouse models of lung adenocarcinoma
Role of RIP4 in lung adenocarcinoma differentiation
Voir toutes les publications (49)
Metabolic plasticity and glucose transporter redundancy dictate lung adenocarcinoma growth
Mutant CTNNB1 and histological heterogeneity define metabolic subtypes of hepatoblastoma
Neutrophils and Snail Orchestrate the Establishment of a Pro-tumor Microenvironment in Lung Cancer
Nonstructural protein 3-4A: the Swiss army knife of hepatitis C virus
Overcoming microenvironmental resistance to PD-1 blockade in genetically engineered lung cancer models
RIP off STAT3 to counteract tumor progression
Regulation of Monocyte Functional Heterogeneity by miR-146a and Relb
Response and resistance to NF-κB inhibitors in mouse models of lung adenocarcinoma
Role of RIP4 in lung adenocarcinoma differentiation
Voir toutes les publications (49)
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