Details
Title
UPMOORE
Formal Name (French)
Unité du Prof. Moore
Formal Name (English)
Prof. Moore Group
Lab Manager
Infoscience team
Group ID
U12001
Affiliated authors
Daniel, Guillaume
Hu, Wanqiu
Moore, Darren
Moser, Roger Philippe
Musso, Alessandra
Podhajska, Agata
Ramonet Jimenez, David
Stafa, Klodjan
Trancikova, Alzbeta
Tsika, Elpida
Hu, Wanqiu
Moore, Darren
Moser, Roger Philippe
Musso, Alessandra
Podhajska, Agata
Ramonet Jimenez, David
Stafa, Klodjan
Trancikova, Alzbeta
Tsika, Elpida
Institute
BMI
Faculty
SV
Note
Members of UPMOORE-unit
Linked resource
http://moorelab.epfl.ch/
Publications
A Rat Model of Progressive Nigral Neurodegeneration Induced by the Parkinson's Disease-Associated G2019S Mutation in LRRK2
Conditional expression of Parkinson's disease-related R1441C LRRK2 in midbrain dopaminergic neurons of mice causes nuclear abnormalities without [...]
Dopaminergic Neuronal Loss, Reduced Neurite Complexity and Autophagic Abnormalities in Transgenic Mice Expressing G2019S Mutant LRRK2
Dopaminergic neurodegeneration induced by Parkinson's disease-linked G2019S LRRK2 is dependent on kinase and GTPase activity
GTPase Activity Plays a Key Role in the Pathobiology of LRRK2
Genetic Mouse Models of Neurodegenerative Diseases
Neurodegenerative phenotypes in an A53T-synuclein transgenic mouse model are independent of LRRK2
Parkin mediates the ubiquitination of VPS35 and modulates retromer-dependent endosomal sorting
Prospects for LLP searches at the LHC in Run 3 and HL-LHC
α-Synuclein in central nervous system and from erythrocytes, mammalian cells, and Escherichia coli exists predominantly as disordered monomer
See complete list of publications (26)
Conditional expression of Parkinson's disease-related R1441C LRRK2 in midbrain dopaminergic neurons of mice causes nuclear abnormalities without [...]
Dopaminergic Neuronal Loss, Reduced Neurite Complexity and Autophagic Abnormalities in Transgenic Mice Expressing G2019S Mutant LRRK2
Dopaminergic neurodegeneration induced by Parkinson's disease-linked G2019S LRRK2 is dependent on kinase and GTPase activity
GTPase Activity Plays a Key Role in the Pathobiology of LRRK2
Genetic Mouse Models of Neurodegenerative Diseases
Neurodegenerative phenotypes in an A53T-synuclein transgenic mouse model are independent of LRRK2
Parkin mediates the ubiquitination of VPS35 and modulates retromer-dependent endosomal sorting
Prospects for LLP searches at the LHC in Run 3 and HL-LHC
α-Synuclein in central nervous system and from erythrocytes, mammalian cells, and Escherichia coli exists predominantly as disordered monomer
See complete list of publications (26)
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