Regulation of arginase pathway in response to wall shear stress.
Alterations of wall shear stress can predispose the endothelium to the development of atherosclerotic plaques. Ample evidence indicates that arginase expression and/or activity correlates with several risk factors for cardiovascular disease including atherosclerosis. We investigated the regulation of arginase pathway in response to distinct patterns of wall shear stress.
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Keywords: Arginase ; Nitric oxide ; Hemodynamic forces ; Wall shear stress ; Oscillatory shear stress ; Mechanotransduction ; Atherosclerosis ; Plaque-Prone Hemodynamics ; Oxide-Mediated Dilation ; Nitric-Oxide ; Endothelial Dysfunction ; Coronary Arterioles ; Cell-Proliferation ; Carotid Arteries ; Smooth-Muscle ; Up-Regulation ; Expression
Record created on 2010-12-16, modified on 2016-08-09