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  4. BAF restricts cGAS on nuclear DNA to prevent innate immune activation
 
research article

BAF restricts cGAS on nuclear DNA to prevent innate immune activation

Guey, Baptiste  
•
Wischnewski, Marilena  
•
Decout, Alexiane  
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August 14, 2020
Science

The appearance of DNA in the cytosol is perceived as a danger signal that stimulates potent immune responses through cyclic guanosine monophosphate–adenosine monophosphate synthase (cGAS). How cells regulate the activity of cGAS toward self-DNA and guard against potentially damaging autoinflammatory responses is a fundamental biological question. Here, we identify barrier-to-autointegration factor 1 (BAF) as a natural opponent of cGAS activity on genomic self-DNA. We show that BAF dynamically outcompetes cGAS for DNA binding, hence prohibiting the formation of DNA-cGAS complexes that are essential for enzymatic activity. Upon acute loss of nuclear membrane integrity, BAF is necessary to restrict cGAS activity on exposed DNA. Our observations reveal a safeguard mechanism, distinct from physical separation, by which cells protect themselves against aberrant immune responses toward genomic DNA.

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Manuscript - BAF restricts cGAS on nuclear DNA.pdf

Type

Postprint

Version

Accepted version

Access type

openaccess

License Condition

CC BY-NC

Size

12.64 MB

Format

Adobe PDF

Checksum (MD5)

79a749c3af002ee2d99ce5038113211a

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